Indiana University Purdue University Indianapolis

Physiology

Physiological research in the Department of Biology is multifaceted with particular strengths in hormonal signaling, eye research, renal research, neural and skeletal development. There is a strong translational component to the research that is enhanced by our proximity to collaborators in the Indiana University School of Medicine on the same campus.  In addition, the Physiology faculty are fortunate to have colleagues in other departments in the School of Science notably Chemistry, Physics and Mathematics who have partnered in research that crosses traditional boundaries and broadens the scope of the possible techniques and approaches that can be used to address important research and medical questions.  With this collective expertise the physiologists in the Department of Biology have been able to conduct studies with direct applicability to understanding and treating diseases such as glaucoma, retinitis pigmentosa, acute kidney injury, polycystic kidney disease, hypertension, metabolic syndrome,  Down Syndrome,  and liver injury.

Faculty:

  • Simon Atkinson, Ph.D.
    Effects of metabolic signaling on cytoskeletal dynamics in kidney injury and disease
  • AJ Baucum
    Identification and characterization of synaptic protein complexes in a brain region called the striatum.
  • Teri Belecky-Adams, Ph.D.
    Aspects of visual system function in health and disease.
  • Bonnie Blazer-Yost, Ph.D.
    Role of kidney, intestinal and lung epithelia in salt and water homeostasis.
  • Gouli Dai, Ph.D.
    Placental hormone-induced maternal hepatic growth response to pregnancy.
  • Jiliang Li, Ph.D.
    Molecular and cellular mechanisms of mechanotransduction, the process of conversion of mechanical signals into biological signals in bone cells, and cell based therapy for bone fracture repair and tissue regeneration.
  • Jason Meyer, Ph.D.
    Induced pluripotent stem cells for studies of neural development and disease.
  • Randall Roper, Ph.D.
    Mechanisms disrupted by trisomy in neural crest and other skeletal precursors in Down Syndrome.